Tsai, I-Ching <br/><br/>
Reactive oxygen species derived from NADPH oxidase 1 and mitochondria mediate angiotensin II-induced smooth muscle cell senescence.  [electronic resource] 

 -  Journal of molecular and cellular cardiology  09 2016 
 - 18-27 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1095-8584 
<br/><br/><label>Standard No.: </label>10.1016/j.yjmcc.2016.07.001  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Angiotensin II--metabolism<br/>Catalase--metabolism<br/>Cells, Cultured<br/>Cellular Senescence<br/>Gene Knockdown Techniques<br/>Humans<br/>Mitochondria, Muscle--drug effects<br/>Muscle, Smooth, Vascular--metabolism<br/>Myocytes, Smooth Muscle--drug effects<br/>NADH, NADPH Oxidoreductases--genetics<br/>NADPH Oxidase 1<br/>NADPH Oxidases--genetics<br/>Oxidation-Reduction<br/>Oxidative Stress<br/>Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha--metabolism<br/>RNA, Small Interfering<br/>Reactive Oxygen Species--metabolism<br/>Uncoupling Protein 2--metabolism