Hui, Kenrie P Y <br/><br/>
Highly pathogenic avian influenza H5N1 virus delays apoptotic responses via activation of STAT3.  [electronic resource] 

 -  Scientific reports  06 2016 
 - 28593 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>2045-2322 
<br/><br/><label>Standard No.: </label>10.1038/srep28593  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Apoptosis--physiology<br/>Birds<br/>Bronchi--metabolism<br/>Caspase 8--metabolism<br/>Caspase 9--metabolism<br/>Cell Line<br/>Cells, Cultured<br/>Cytokines--metabolism<br/>Dogs<br/>Epithelial Cells--metabolism<br/>Humans<br/>Inflammation--metabolism<br/>Influenza A Virus, H1N1 Subtype--pathogenicity<br/>Influenza A Virus, H5N1 Subtype--pathogenicity<br/>Influenza in Birds--metabolism<br/>Influenza, Human--metabolism<br/>Madin Darby Canine Kidney Cells<br/>Orthomyxoviridae Infections<br/>STAT3 Transcription Factor--metabolism<br/>Virus Replication--physiology