Lu, Jingchen <br/><br/>
EBV-LMP1 suppresses the DNA damage response through DNA-PK/AMPK signaling to promote radioresistance in nasopharyngeal carcinoma.  [electronic resource] 

 -  Cancer letters  09 2016 
 - 191-200 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>1872-7980 
<br/><br/><label>Standard No.: </label>10.1016/j.canlet.2016.05.032  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>AMP-Activated Protein Kinases--metabolism<br/>Animals<br/>Apoptosis--radiation effects<br/>Carcinoma--enzymology<br/>Cell Line, Tumor<br/>DNA Breaks, Double-Stranded<br/>DNA Repair<br/>DNA-Activated Protein Kinase--metabolism<br/>Dose-Response Relationship, Radiation<br/>Enzyme Activation<br/>Epstein-Barr Virus Infections--virology<br/>Female<br/>Glycolysis<br/>Herpesvirus 4, Human--metabolism<br/>Host-Pathogen Interactions<br/>Humans<br/>Mice, Inbred BALB C<br/>Mice, Nude<br/>Nasopharyngeal Carcinoma<br/>Nasopharyngeal Neoplasms--enzymology<br/>Nuclear Proteins--metabolism<br/>Phosphorylation<br/>Radiation Tolerance<br/>Signal Transduction--radiation effects<br/>Time Factors<br/>Tumor Burden--radiation effects<br/>Viral Matrix Proteins--metabolism<br/>Xenograft Model Antitumor Assays