Chen, Bin <br/><br/>
Blocking GluN2B subunits reverses the enhanced seizure susceptibility after prolonged febrile seizures with a wide therapeutic time-window.  [electronic resource] 

 -  Experimental neurology  09 2016 
 - 29-38 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1090-2430 
<br/><br/><label>Standard No.: </label>10.1016/j.expneurol.2016.05.034  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Animals, Newborn<br/>Disease Models, Animal<br/>Disease Susceptibility--metabolism<br/>Electroshock--adverse effects<br/>Excitatory Amino Acid Agonists--toxicity<br/>Excitatory Amino Acid Antagonists--therapeutic use<br/>Exploratory Behavior--drug effects<br/>Humans<br/>Interleukin 1 Receptor Antagonist Protein--therapeutic use<br/>Interleukin-1beta--therapeutic use<br/>Kainic Acid--toxicity<br/>Maze Learning--drug effects<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Knockout<br/>Piperidines--therapeutic use<br/>Rats<br/>Rats, Sprague-Dawley<br/>Receptors, Interleukin-1 Type I--genetics<br/>Receptors, N-Methyl-D-Aspartate--metabolism<br/>Seizures, Febrile--etiology