Marks, William D <br/><br/>
HIV-1 Tat causes cognitive deficits and selective loss of parvalbumin, somatostatin, and neuronal nitric oxide synthase expressing hippocampal CA1 interneuron subpopulations.  [electronic resource] 

 -  Journal of neurovirology  12 2016 
 - 747-762 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural 
<br/><br/><label>ISSN: </label>1538-2443 
<br/><br/><label>Standard No.: </label>10.1007/s13365-016-0447-2  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>CA1 Region, Hippocampal--metabolism<br/>Cognition--physiology<br/>Cognitive Dysfunction--genetics<br/>Gene Expression Regulation<br/>Interneurons--metabolism<br/>Male<br/>Maze Learning<br/>Mice<br/>Mice, Transgenic<br/>Neuropeptide Y--genetics<br/>Nitric Oxide Synthase Type I--deficiency<br/>Parvalbumins--deficiency<br/>Signal Transduction<br/>Somatostatin--deficiency<br/>Transgenes<br/>tat Gene Products, Human Immunodeficiency Virus--genetics