Newton, K <br/><br/>
RIPK3 deficiency or catalytically inactive RIPK1 provides greater benefit than MLKL deficiency in mouse models of inflammation and tissue injury.  [electronic resource] 

 -  Cell death and differentiation  09 2016 
 - 1565-76 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>1476-5403 
<br/><br/><label>Standard No.: </label>10.1038/cdd.2016.46  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Apoptosis--drug effects<br/>Ceruletide--toxicity<br/>Colitis--chemically induced<br/>Dextran Sulfate--toxicity<br/>Disease Models, Animal<br/>Female<br/>Inflammation--metabolism<br/>Lipopolysaccharides--toxicity<br/>Male<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Knockout<br/>Pancreatitis--chemically induced<br/>Protein Kinases--deficiency<br/>Receptor-Interacting Protein Serine-Threonine Kinases--antagonists & inhibitors<br/>Reperfusion Injury--metabolism<br/>Sepsis--etiology<br/>Systemic Inflammatory Response Syndrome--etiology<br/>Tumor Necrosis Factor alpha-Induced Protein 3--deficiency