Chuang, Gin C <br/><br/>
Environmentally Persistent Free Radicals Cause Apoptosis in HL-1 Cardiomyocytes.  [electronic resource] 

 -  Cardiovascular toxicology  04 2017 
 - 140-149 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural 
<br/><br/><label>ISSN: </label>1559-0259 
<br/><br/><label>Standard No.: </label>10.1007/s12012-016-9367-x  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Antioxidants--pharmacology<br/>Apoptosis--drug effects<br/>Caspases--metabolism<br/>Cell Line<br/>Dose-Response Relationship, Drug<br/>Environmental Pollutants--toxicity<br/>Free Radicals--toxicity<br/>Male<br/>Membrane Potential, Mitochondrial--drug effects<br/>Mice<br/>Mitochondria--drug effects<br/>Myocytes, Cardiac--drug effects<br/>Oxidative Stress--drug effects<br/>Particulate Matter--toxicity<br/>Poly(ADP-ribose) Polymerases--metabolism<br/>Rats<br/>Rats, Sprague-Dawley<br/>Signal Transduction--drug effects<br/>Time Factors