Tse, Kai-Hei <br/><br/>
PGE2 released by primary sensory neurons modulates Toll-like receptor 4 activities through an EP4 receptor-dependent process.  [electronic resource] 

 -  Journal of neuroimmunology  Apr 2016 
 - 8-16 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1872-8421 
<br/><br/><label>Standard No.: </label>10.1016/j.jneuroim.2016.02.005  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Cells, Cultured<br/>Culture Media, Conditioned--pharmacology<br/>Cyclic AMP--metabolism<br/>Cyclic AMP-Dependent Protein Kinases--metabolism<br/>Cyclooxygenase 2--genetics<br/>Dinoprostone--metabolism<br/>Enzyme Inhibitors--pharmacology<br/>Ganglia, Spinal--cytology<br/>Gene Expression Regulation--drug effects<br/>Glial Fibrillary Acidic Protein--metabolism<br/>Lipopolysaccharide Receptors--metabolism<br/>Lipopolysaccharides--pharmacology<br/>Male<br/>Quinolines--pharmacology<br/>Rats<br/>Rats, Sprague-Dawley<br/>Receptors, Prostaglandin E, EP4 Subtype--antagonists & inhibitors<br/>Sensory Receptor Cells--drug effects<br/>Sulfonamides--pharmacology<br/>Thiophenes--pharmacology<br/>Toll-Like Receptor 4--metabolism<br/>Triazoles--pharmacology<br/>Tumor Necrosis Factor-alpha--genetics