Leng, Chao <br/><br/>
An integrin beta4-EGFR unit promotes hepatocellular carcinoma lung metastases by enhancing anchorage independence through activation of FAK-AKT pathway.  [electronic resource] 

 -  Cancer letters  06 2016 
 - 188-96 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1872-7980 
<br/><br/><label>Standard No.: </label>10.1016/j.canlet.2016.03.023  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adult<br/>Aged<br/>Animals<br/>Anoikis<br/>Carcinoma, Hepatocellular--enzymology<br/>Cell Proliferation<br/>Enzyme Activation<br/>ErbB Receptors--metabolism<br/>Female<br/>Focal Adhesion Kinase 1--metabolism<br/>Gene Expression Regulation, Neoplastic<br/>Hep G2 Cells<br/>Heterografts<br/>Humans<br/>Integrin beta4--genetics<br/>Liver Neoplasms--enzymology<br/>Lung Neoplasms--enzymology<br/>Male<br/>Mice, Inbred BALB C<br/>Mice, Nude<br/>Middle Aged<br/>Neoplasm Transplantation<br/>Proto-Oncogene Proteins c-akt--genetics<br/>RNA Interference<br/>Signal Transduction<br/>Transfection<br/>Tumor Burden