Fujimura, Masatake <br/><br/>
Prenatal low-dose methylmercury exposure impairs neurite outgrowth and synaptic protein expression and suppresses TrkA pathway activity and eEF1A1 expression in the rat cerebellum.  [electronic resource] 

 -  Toxicology and applied pharmacology  May 2016 
 - 1-8 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1096-0333 
<br/><br/><label>Standard No.: </label>10.1016/j.taap.2016.03.002  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Blotting, Western<br/>Cerebellum--drug effects<br/>Dose-Response Relationship, Drug<br/>Down-Regulation<br/>Environmental Pollutants--toxicity<br/>Female<br/>Male<br/>Methylmercury Compounds--toxicity<br/>Neurites--drug effects<br/>PC12 Cells<br/>Peptide Elongation Factor 1--genetics<br/>Pregnancy<br/>Prenatal Exposure Delayed Effects--chemically induced<br/>Rats<br/>Rats, Wistar<br/>Receptor, trkA--antagonists & inhibitors<br/>Synapses--drug effects