Fan, Tianli <br/><br/>
Bit1 knockdown contributes to growth suppression as well as the decreases of migration and invasion abilities in esophageal squamous cell carcinoma via suppressing FAK-paxillin pathway.  [electronic resource] 

 -  Molecular cancer  Mar 2016 
 - 23 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1476-4598 
<br/><br/><label>Standard No.: </label>10.1186/s12943-016-0507-5  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Apoptosis<br/>Carboxylic Ester Hydrolases--metabolism<br/>Carcinogenesis--pathology<br/>Carcinoma, Squamous Cell--pathology<br/>Cell Line, Tumor<br/>Cell Movement<br/>Cell Proliferation<br/>Down-Regulation<br/>Esophageal Neoplasms--pathology<br/>Esophageal Squamous Cell Carcinoma<br/>Focal Adhesion Protein-Tyrosine Kinases--metabolism<br/>Gene Knockdown Techniques<br/>Humans<br/>Matrix Metalloproteinase 2--metabolism<br/>Mice, Nude<br/>Mitochondrial Proteins--metabolism<br/>Neoplasm Invasiveness<br/>Oligonucleotide Array Sequence Analysis<br/>Paxillin--metabolism<br/>Protein Binding<br/>Proto-Oncogene Proteins c-bcl-2--metabolism<br/>RNA, Small Interfering--metabolism<br/>Signal Transduction<br/>Xenograft Model Antitumor Assays