Gu, Hongmei <br/><br/>
Contribution of the anaphylatoxin receptors, C3aR and C5aR, to the pathogenesis of pulmonary fibrosis.  [electronic resource] 

 -  FASEB journal : official publication of the Federation of American Societies for Experimental Biology  06 2016 
 - 2336-50 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>1530-6860 
<br/><br/><label>Standard No.: </label>10.1096/fj.201500044  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Aged<br/>Aged, 80 and over<br/>Animals<br/>Antibiotics, Antineoplastic--toxicity<br/>Bleomycin--toxicity<br/>Cell Line<br/>Collagen Type I, alpha 1 Chain<br/>Complement Membrane Attack Complex--genetics<br/>Down-Regulation<br/>Fibroblasts--metabolism<br/>Gene Expression Regulation--physiology<br/>Humans<br/>Lung Injury--chemically induced<br/>Mice<br/>Mice, Inbred C57BL<br/>Middle Aged<br/>Pulmonary Fibrosis--chemically induced<br/>RNA Interference<br/>Receptor, Anaphylatoxin C5a--genetics<br/>Receptors, Complement--genetics<br/>Signal Transduction--physiology<br/>Transforming Growth Factor beta--genetics<br/>Up-Regulation