Antigny, Fabrice <br/><br/>
Potassium Channel Subfamily K Member 3 (KCNK3) Contributes to the Development of Pulmonary Arterial Hypertension.  [electronic resource] 

 -  Circulation  Apr 2016 
 - 1371-85 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1524-4539 
<br/><br/><label>Standard No.: </label>10.1161/CIRCULATIONAHA.115.020951  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adventitia--pathology<br/>Animals<br/>Bone Morphogenetic Protein Receptors, Type II--genetics<br/>Cell Division<br/>Endothelium, Vascular--pathology<br/>Fibroblasts--pathology<br/>Genetic Predisposition to Disease<br/>Hemodynamics<br/>Humans<br/>Hypertension, Pulmonary--chemically induced<br/>Hypertrophy, Right Ventricular--etiology<br/>Inflammation<br/>Male<br/>Membrane Potentials<br/>Monocrotaline--toxicity<br/>Mutation<br/>Myocytes, Smooth Muscle--pathology<br/>Nerve Tissue Proteins--antagonists & inhibitors<br/>Patch-Clamp Techniques<br/>Potassium Channels, Tandem Pore Domain--antagonists & inhibitors<br/>Rats<br/>Rats, Sprague-Dawley<br/>Rats, Wistar<br/>Sulfonamides--pharmacology<br/>Vascular Resistance<br/>ortho-Aminobenzoates--pharmacology