Biocompatible arsenic trioxide nanoparticles induce cell cycle arrest by p21(WAF1/CIP1) expression via epigenetic remodeling in LNCaP and PC3 cell lines. [electronic resource]
- Life sciences Mar 2016
- 41-52 p. digital
Publication Type: Journal Article
1879-0631
10.1016/j.lfs.2016.02.042 doi
Antineoplastic Agents--pharmacology Arsenic Trioxide Arsenicals--pharmacology Biocompatible Materials--pharmacology Cell Cycle Checkpoints--drug effects Cell Line, Tumor Cyclin-Dependent Kinase Inhibitor p21--biosynthesis Dose-Response Relationship, Drug Epigenesis, Genetic--drug effects Humans Male Metal Nanoparticles--therapeutic use Oxides--pharmacology Prostatic Neoplasms--drug therapy