Wai, Timothy <br/><br/>
Imbalanced OPA1 processing and mitochondrial fragmentation cause heart failure in mice.  [electronic resource] 

 -  Science (New York, N.Y.)  Dec 2015 
 - aad0116 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1095-9203 
<br/><br/><label>Standard No.: </label>10.1126/science.aad0116  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Cardiomyopathy, Dilated--genetics<br/>Diet, High-Fat<br/>Embryonic Development<br/>Female<br/>GTP Phosphohydrolases<br/>Gene Deletion<br/>Heart--embryology<br/>Heart Failure--genetics<br/>Male<br/>Metalloendopeptidases--genetics<br/>Metalloproteases--genetics<br/>Mice<br/>Mice, Knockout<br/>Mitochondria, Heart--metabolism<br/>Mitochondrial Dynamics<br/>Mitochondrial Proteins--genetics<br/>Mitophagy<br/>Muscle, Skeletal--enzymology<br/>Myocardium--metabolism<br/>Myocytes, Cardiac--enzymology<br/>Proteolysis