Li, Haiying <br/><br/>
Acute Exercise-Induced Mitochondrial Stress Triggers an Inflammatory Response in the Myocardium via NLRP3 Inflammasome Activation with Mitophagy.  [electronic resource] 

 -  Oxidative medicine and cellular longevity  2016 
 - 1987149 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1942-0994 
<br/><br/><label>Standard No.: </label>10.1155/2016/1987149  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Antioxidants--pharmacology<br/>Autophagy--drug effects<br/>Carrier Proteins--metabolism<br/>Cell Respiration--drug effects<br/>Inflammasomes--drug effects<br/>Inflammation--pathology<br/>Interleukin-1beta--metabolism<br/>Male<br/>Membrane Potential, Mitochondrial--drug effects<br/>Mitochondrial Proton-Translocating ATPases--metabolism<br/>Mitophagy--drug effects<br/>Myocardium--metabolism<br/>NLR Family, Pyrin Domain-Containing 3 Protein<br/>Oxidants--toxicity<br/>Oxidative Stress--drug effects<br/>Physical Conditioning, Animal<br/>Rats, Sprague-Dawley<br/>Stress, Physiological--drug effects