The Pathogenic Mutation T182A Converts the Prion Protein into a Molten Globule-like Conformation Whose Misfolding to Oligomers but Not to Fibrils Is Drastically Accelerated. [electronic resource]
- Biochemistry Jan 2016
- 459-69 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1520-4995
10.1021/acs.biochem.5b01266 doi
Amyloid--chemistry Animals Hydrogen-Ion Concentration Mice Mutation Prions--chemistry Protein Folding Protein Structure, Secondary Protein Structure, Tertiary Thermodynamics