Gomes Fernandes, Maria <br/><br/>
BMP-SMAD Signaling Regulates Lineage Priming, but Is Dispensable for Self-Renewal in Mouse Embryonic Stem Cells.  [electronic resource] 

 -  Stem cell reports  Jan 2016 
 - 85-94 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>2213-6711 
<br/><br/><label>Standard No.: </label>10.1016/j.stemcr.2015.11.012  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Bone Morphogenetic Proteins--genetics<br/>Cell Lineage--genetics<br/>Cell Self Renewal--genetics<br/>Cells, Cultured<br/>DNA (Cytosine-5-)-Methyltransferases--genetics<br/>DNA Methylation--genetics<br/>DNA-Binding Proteins--genetics<br/>Dioxygenases<br/>Gene Expression Profiling--methods<br/>Mice<br/>Mice, Knockout<br/>Mice, Transgenic<br/>Microscopy, Confocal<br/>Mouse Embryonic Stem Cells--cytology<br/>Pluripotent Stem Cells--cytology<br/>Proto-Oncogene Proteins--genetics<br/>Reverse Transcriptase Polymerase Chain Reaction<br/>Signal Transduction--genetics<br/>Smad Proteins, Receptor-Regulated--genetics<br/>Smad1 Protein--genetics<br/>Smad5 Protein--genetics