Elrick, Mollisa M <br/><br/>
Neuronostatin acts via GPR107 to increase cAMP-independent PKA phosphorylation and proglucagon mRNA accumulation in pancreatic α-cells.  [electronic resource] 

 -  American journal of physiology. Regulatory, integrative and comparative physiology  Jan 2016 
 - R143-55 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural 
<br/><br/><label>ISSN: </label>1522-1490 
<br/><br/><label>Standard No.: </label>10.1152/ajpregu.00369.2014  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Cell Line<br/>Cyclic AMP--metabolism<br/>Cyclic AMP-Dependent Protein Kinases--metabolism<br/>Glucagon-Secreting Cells--drug effects<br/>Humans<br/>Male<br/>Mice<br/>Peptide Fragments--metabolism<br/>Peptide Hormones--metabolism<br/>Phosphorylation<br/>Proglucagon--genetics<br/>RNA Interference<br/>RNA, Messenger--genetics<br/>Rats, Sprague-Dawley<br/>Receptors, G-Protein-Coupled--agonists<br/>Signal Transduction--drug effects<br/>Somatostatin--metabolism<br/>Transfection<br/>Up-Regulation