Cai, Jingjing <br/><br/>
HMGB1-Driven Inflammation and Intimal Hyperplasia After Arterial Injury Involves Cell-Specific Actions Mediated by TLR4.  [electronic resource] 

 -  Arteriosclerosis, thrombosis, and vascular biology  Dec 2015 
 - 2579-93 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1524-4636 
<br/><br/><label>Standard No.: </label>10.1161/ATVBAHA.115.305789  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adaptor Proteins, Vesicular Transport--metabolism<br/>Animals<br/>Carotid Arteries--metabolism<br/>Carotid Artery Injuries--genetics<br/>Cell Movement<br/>Cell Proliferation<br/>Cells, Cultured<br/>Chemotaxis, Leukocyte<br/>Cytokines--metabolism<br/>Disease Models, Animal<br/>HMGB1 Protein--deficiency<br/>Humans<br/>Hyperplasia<br/>Inflammation Mediators--metabolism<br/>Macrophages, Peritoneal--metabolism<br/>Mice, Inbred C57BL<br/>Mice, Knockout<br/>Myeloid Differentiation Factor 88--genetics<br/>Myocytes, Smooth Muscle--metabolism<br/>Neointima<br/>Signal Transduction<br/>Time Factors<br/>Toll-Like Receptor 4--deficiency<br/>Vascular System Injuries--genetics<br/>Vasculitis--genetics