Cao, Jing <br/><br/>
Overproduction of reactive oxygen species and activation of MAPKs are involved in apoptosis induced by PM2.5 in rat cardiac H9c2 cells.  [electronic resource] 

 -  Journal of applied toxicology : JAT  Apr 2016 
 - 609-17 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1099-1263 
<br/><br/><label>Standard No.: </label>10.1002/jat.3249  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Apoptosis--drug effects<br/>Caspase 3--genetics<br/>Cell Line<br/>Cell Survival--drug effects<br/>Enzyme Inhibitors--pharmacology<br/>Extracellular Signal-Regulated MAP Kinases--antagonists & inhibitors<br/>Flavonoids--pharmacology<br/>JNK Mitogen-Activated Protein Kinases--genetics<br/>Myocytes, Cardiac--drug effects<br/>Oxidative Stress--drug effects<br/>Particulate Matter--toxicity<br/>Phosphorylation<br/>Rats<br/>Reactive Oxygen Species--metabolism<br/>Up-Regulation<br/>p38 Mitogen-Activated Protein Kinases--antagonists & inhibitors