Amaral, Flávio A <br/><br/>
Transmembrane TNF-α is sufficient for articular inflammation and hypernociception in a mouse model of gout.  [electronic resource] 

 -  European journal of immunology  Jan 2016 
 - 204-11 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1521-4141 
<br/><br/><label>Standard No.: </label>10.1002/eji.201545798  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Disease Models, Animal<br/>Gout--complications<br/>Hyperalgesia--etiology<br/>Inflammation--immunology<br/>Interleukin-1beta--immunology<br/>Knee Joint<br/>Male<br/>Membrane Proteins--metabolism<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Transgenic<br/>Physical Stimulation<br/>Real-Time Polymerase Chain Reaction<br/>Tumor Necrosis Factor-alpha--metabolism<br/>Uric Acid--adverse effects