Lee, Youjin <br/><br/>
IL-21R signaling is critical for induction of spontaneous experimental autoimmune encephalomyelitis.  [electronic resource] 

 -  The Journal of clinical investigation  Nov 2015 
 - 4011-20 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1558-8238 
<br/><br/><label>Standard No.: </label>10.1172/JCI75933  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Antigen Presentation<br/>Cells, Cultured<br/>Disease Susceptibility<br/>Encephalomyelitis, Autoimmune, Experimental--immunology<br/>Interferon-gamma--biosynthesis<br/>Interleukin-21 Receptor alpha Subunit--deficiency<br/>Interleukins--physiology<br/>Lymphocyte Activation<br/>Lymphopoiesis<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Knockout<br/>Mice, Transgenic<br/>Myelin-Oligodendrocyte Glycoprotein--immunology<br/>Peptide Fragments--immunology<br/>Receptors, Interleukin--biosynthesis<br/>Signal Transduction<br/>Specific Pathogen-Free Organisms<br/>Th17 Cells--immunology<br/>Interleukin-21