Liu, Su
Wnt/Ryk signaling contributes to neuropathic pain by regulating sensory neuron excitability and spinal synaptic plasticity in rats. [electronic resource]
- Pain Dec 2015
- 2572-2584 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
ISSN: 1872-6623
Standard No.: 10.1097/j.pain.0000000000000366 doi
Subjects--Topical Terms: Animals Astrocytes--metabolism Behavior, Animal--drug effects Calcium Signaling Calcium-Calmodulin-Dependent Protein Kinase Type 2--drug effects Cyclic AMP Response Element-Binding Protein--drug effects Ganglia, Spinal--metabolism Hyperalgesia--genetics Locomotion--drug effects MAP Kinase Signaling System--drug effects Male Nerve Fibers, Unmyelinated--drug effects Neuralgia--genetics Neuronal Plasticity--drug effects Pain Threshold--drug effects Patch-Clamp Techniques Posterior Horn Cells--metabolism Protein Kinase C Rats Rats, Sprague-Dawley Real-Time Polymerase Chain Reaction Receptor Protein-Tyrosine Kinases--antagonists & inhibitors Receptors, N-Methyl-D-Aspartate--drug effects Sciatic Nerve--injuries Sensory Receptor Cells--metabolism Signal Transduction Spinal Cord--metabolism Wnt Proteins--antagonists & inhibitors Wnt Signaling Pathway--drug effects Wnt3A Protein--antagonists & inhibitors src-Family Kinases--drug effects