Saigusa, R <br/><br/>
Fli1 deficiency contributes to the downregulation of endothelial protein C receptor in systemic sclerosis: a possible role in prothrombotic conditions.  [electronic resource] 

 -  The British journal of dermatology  Feb 2016 
 - 338-47 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1365-2133 
<br/><br/><label>Standard No.: </label>10.1111/bjd.14183  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adult<br/>Aged<br/>Analysis of Variance<br/>Animals<br/>Antigens, CD--physiology<br/>Bleomycin--pharmacology<br/>Bosentan<br/>Case-Control Studies<br/>Cells, Cultured<br/>Down-Regulation--physiology<br/>Endothelial Cells--metabolism<br/>Endothelial Protein C Receptor<br/>Endothelin Receptor Antagonists--pharmacology<br/>Endothelium, Vascular--metabolism<br/>Enzyme-Linked Immunosorbent Assay<br/>Female<br/>Fibrinolysin--drug effects<br/>Humans<br/>Male<br/>Mice<br/>Microvessels--metabolism<br/>Middle Aged<br/>Proto-Oncogene Protein c-fli-1--deficiency<br/>Receptors, Cell Surface--physiology<br/>Scleroderma, Systemic--blood<br/>Sulfonamides--pharmacology<br/>Thrombophilia--etiology<br/>alpha-2-Antiplasmin--drug effects