Parajuli, Bijay <br/><br/>
CCL11 enhances excitotoxic neuronal death by producing reactive oxygen species in microglia.  [electronic resource] 

 -  Glia  Dec 2015 
 - 2274-84 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1098-1136 
<br/><br/><label>Standard No.: </label>10.1002/glia.22892  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Astrocytes--physiology<br/>CD11b Antigen--metabolism<br/>Cell Death--physiology<br/>Cell Movement--physiology<br/>Cell Survival--physiology<br/>Cells, Cultured<br/>Chemokine CCL11--metabolism<br/>Coculture Techniques<br/>DNA-Binding Proteins<br/>Glutamic Acid--toxicity<br/>Mice, Inbred C57BL<br/>Microglia--metabolism<br/>Microtubule-Associated Proteins--metabolism<br/>NADH, NADPH Oxidoreductases--metabolism<br/>NADPH Oxidase 1<br/>Nerve Tissue Proteins--metabolism<br/>Neurons--physiology<br/>Nuclear Proteins--metabolism<br/>RNA, Messenger--metabolism<br/>Reactive Oxygen Species--metabolism<br/>Recombinant Proteins--metabolism