Li, Wei <br/><br/>
Tanshinone IIA represses inflammatory response and reduces radiculopathic pain by inhibiting IRAK-1 and NF-κB/p38/JNK signaling.  [electronic resource] 

 -  International immunopharmacology  Sep 2015 
 - 382-9 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>1878-1705 
<br/><br/><label>Standard No.: </label>10.1016/j.intimp.2015.06.032  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Abietanes--pharmacology<br/>Analgesics--pharmacology<br/>Animals<br/>Anti-Inflammatory Agents, Non-Steroidal--pharmacology<br/>Cell Survival--drug effects<br/>Cells, Cultured<br/>Cyclooxygenase 2--genetics<br/>Drugs, Chinese Herbal--pharmacology<br/>Female<br/>Humans<br/>Interleukin-1 Receptor-Associated Kinases--metabolism<br/>Interleukin-1beta<br/>Intervertebral Disc--cytology<br/>Intervertebral Disc Degeneration--drug therapy<br/>Intervertebral Disc Displacement--drug therapy<br/>JNK Mitogen-Activated Protein Kinases--metabolism<br/>Matrix Metalloproteinase 1--genetics<br/>Matrix Metalloproteinase 13--genetics<br/>NF-kappa B--metabolism<br/>Nitric Oxide Synthase Type II--genetics<br/>Pain--drug therapy<br/>Rats, Sprague-Dawley<br/>p38 Mitogen-Activated Protein Kinases--metabolism