Wu, Dan <br/><br/>
CTRP3 attenuates post-infarct cardiac fibrosis by targeting Smad3 activation and inhibiting myofibroblast differentiation.  [electronic resource] 

 -  Journal of molecular medicine (Berlin, Germany)  Dec 2015 
 - 1311-25 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1432-1440 
<br/><br/><label>Standard No.: </label>10.1007/s00109-015-1309-8  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>AMP-Activated Protein Kinases--metabolism<br/>Adenoviridae--genetics<br/>Adipokines--genetics<br/>Animals<br/>Cardiomegaly--etiology<br/>Cell Differentiation--drug effects<br/>Cells, Cultured<br/>Disease Models, Animal<br/>E1A-Associated p300 Protein--metabolism<br/>Fibrosis<br/>Gene Expression<br/>Gene Expression Regulation--drug effects<br/>Gene Knockdown Techniques<br/>Gene Transfer Techniques<br/>Genetic Vectors--administration & dosage<br/>Male<br/>Myocardial Infarction--complications<br/>Myofibroblasts--cytology<br/>Phosphorylation<br/>Protein Binding<br/>Protein Transport<br/>Proto-Oncogene Proteins c-akt--metabolism<br/>Rats<br/>Smad3 Protein--metabolism<br/>Transduction, Genetic<br/>Transforming Growth Factor beta1--metabolism<br/>Ventricular Remodeling--genetics