Wu, Haodi <br/><br/>
Epigenetic Regulation of Phosphodiesterases 2A and 3A Underlies Compromised β-Adrenergic Signaling in an iPSC Model of Dilated Cardiomyopathy.  [electronic resource] 

 -  Cell stem cell  Jul 2015 
 - 89-100 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1875-9777 
<br/><br/><label>Standard No.: </label>10.1016/j.stem.2015.04.020  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adrenergic beta-Agonists--pharmacology<br/>Cardiomyopathy, Dilated--genetics<br/>Cell Differentiation<br/>Cells, Cultured<br/>Cyclic Nucleotide Phosphodiesterases, Type 2--genetics<br/>Cyclic Nucleotide Phosphodiesterases, Type 3--genetics<br/>Epigenesis, Genetic<br/>Heart Rate--drug effects<br/>Humans<br/>Induced Pluripotent Stem Cells--drug effects<br/>Isoproterenol--pharmacology<br/>Models, Cardiovascular<br/>Mutant Proteins--genetics<br/>Mutation<br/>Myocardial Contraction--drug effects<br/>Myocytes, Cardiac--drug effects<br/>Receptors, Adrenergic, beta--metabolism<br/>Signal Transduction<br/>Troponin T--genetics<br/>Up-Regulation