Szlachcic, Wojciech J <br/><br/>
Huntington disease iPSCs show early molecular changes in intracellular signaling, the expression of oxidative stress proteins and the p53 pathway.  [electronic resource] 

 -  Disease models & mechanisms  Sep 2015 
 - 1047-57 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1754-8411 
<br/><br/><label>Standard No.: </label>10.1242/dmm.019406  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Base Sequence<br/>Cell Differentiation<br/>Child<br/>Disease Models, Animal<br/>Embryonic Stem Cells--cytology<br/>Enzyme Activation<br/>Humans<br/>Huntingtin Protein<br/>Huntington Disease--metabolism<br/>Induced Pluripotent Stem Cells--cytology<br/>MAP Kinase Signaling System<br/>Mice<br/>Molecular Sequence Data<br/>Mutation<br/>Nerve Tissue Proteins--genetics<br/>Nuclear Proteins--genetics<br/>Oxidative Stress<br/>Phosphorylation<br/>Signal Transduction<br/>Superoxide Dismutase--metabolism<br/>Superoxide Dismutase-1<br/>Tumor Suppressor Protein p53--metabolism<br/>Wnt Proteins--metabolism<br/>Young Adult<br/>beta Catenin--metabolism