Pekarova, Michaela <br/><br/>
Asymmetric dimethyl arginine induces pulmonary vascular dysfunction via activation of signal transducer and activator of transcription 3 and stabilization of hypoxia-inducible factor 1-alpha.  [electronic resource] 

 -  Vascular pharmacology  Oct 2015 
 - 138-48 p.  digital 
<br/><br/> Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1879-3649 
<br/><br/><label>Standard No.: </label>10.1016/j.vph.2015.06.005  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Amidohydrolases--metabolism<br/>Arginine--analogs & derivatives<br/>Calcium--metabolism<br/>Cell Hypoxia<br/>Cell Proliferation--drug effects<br/>Cells, Cultured<br/>Dose-Response Relationship, Drug<br/>Endothelial Cells--drug effects<br/>Humans<br/>Hypertension, Pulmonary--etiology<br/>Hypoxia-Inducible Factor 1, alpha Subunit--metabolism<br/>Inflammation Mediators--metabolism<br/>Intercellular Adhesion Molecule-1--metabolism<br/>Muscle, Smooth, Vascular--drug effects<br/>Myocytes, Smooth Muscle--drug effects<br/>Nitric Oxide--metabolism<br/>Nitric Oxide Synthase Type III--metabolism<br/>Pulmonary Artery--drug effects<br/>STAT3 Transcription Factor--metabolism<br/>Signal Transduction--drug effects