Kikuchi, Hidehiko <br/><br/>
Lack of GCN5 remarkably enhances the resistance against prolonged endoplasmic reticulum stress-induced apoptosis through up-regulation of Bcl-2 gene expression.  [electronic resource] 

 -  Biochemical and biophysical research communications  Aug 2015 
 - 870-5 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1090-2104 
<br/><br/><label>Standard No.: </label>10.1016/j.bbrc.2015.06.027  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Apoptosis--drug effects<br/>Cell Line<br/>Chickens<br/>Endoplasmic Reticulum--drug effects<br/>Genes, bcl-2<br/>Histone Acetyltransferases--genetics<br/>Thapsigargin--pharmacology<br/>Up-Regulation