Wang, Wei <br/><br/>
Inhibition of microglial activation contributes to propofol-induced protection against post-cardiac arrest brain injury in rats.  [electronic resource] 

 -  Journal of neurochemistry  Sep 2015 
 - 892-903 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1471-4159 
<br/><br/><label>Standard No.: </label>10.1111/jnc.13179  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adenosine Triphosphate--physiology<br/>Animals<br/>Asphyxia--complications<br/>Brain Ischemia--etiology<br/>CA1 Region, Hippocampal--drug effects<br/>CD11b Antigen--biosynthesis<br/>Cardiopulmonary Resuscitation<br/>Cells, Cultured<br/>Heart Arrest--complications<br/>Interleukin-1beta--metabolism<br/>MAP Kinase Signaling System--drug effects<br/>Male<br/>Maze Learning<br/>Mice<br/>Microglia--drug effects<br/>Minocycline--pharmacology<br/>Models, Animal<br/>Nerve Tissue Proteins--biosynthesis<br/>Neuroprotective Agents--pharmacology<br/>Phosphorylation<br/>Propofol--pharmacology<br/>Protein Processing, Post-Translational<br/>Random Allocation<br/>Rats<br/>Rats, Sprague-Dawley<br/>Receptors, Purinergic P2X7--biosynthesis<br/>Tumor Necrosis Factor-alpha--metabolism<br/>p38 Mitogen-Activated Protein Kinases--metabolism