Stefano, George B <br/><br/>
Hypoxia defined as a common culprit/initiation factor in mitochondrial-mediated proinflammatory processes.  [electronic resource] 

 -  Medical science monitor : international medical journal of experimental and clinical research  May 2015 
 - 1478-84 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Review 
<br/><br/><label>ISSN: </label>1643-3750 
<br/><br/><label>Standard No.: </label>10.12659/MSM.894437  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adenosine Triphosphate--biosynthesis<br/>Animals<br/>Biological Evolution<br/>Cell Hypoxia--physiology<br/>Cellular Microenvironment<br/>Cellular Senescence<br/>Cytokines--metabolism<br/>Energy Metabolism<br/>Gene Expression Regulation<br/>Humans<br/>Immunity, Innate<br/>Immunocompetence<br/>Inflammation--etiology<br/>Invertebrates--physiology<br/>Microglia--physiology<br/>Mitochondria--physiology<br/>Models, Biological<br/>Neuroimmunomodulation--physiology<br/>Nitric Oxide--physiology<br/>Nitrites--metabolism<br/>Opioid Peptides--metabolism<br/>Signal Transduction<br/>Vertebrates--physiology