Alterations in left ventricular function during intermittent hypoxia: Possible involvement of O-GlcNAc protein and MAPK signaling. [electronic resource]
- International journal of molecular medicine Jul 2015
- 150-8 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1791-244X
10.3892/ijmm.2015.2198 doi
Acetylglucosamine--metabolism Animals Apoptosis--physiology Blood Gas Analysis Blood Pressure--physiology Calcium-Calmodulin-Dependent Protein Kinase Type 2--metabolism Cardiovascular Diseases--physiopathology Caspase 3--metabolism Cell Hypoxia--physiology Extracellular Signal-Regulated MAP Kinases--metabolism In Situ Nick-End Labeling Interleukin-6--metabolism MAP Kinase Signaling System--physiology Male Myocardium--metabolism Myocytes, Cardiac--metabolism N-Acetylglucosaminyltransferases--metabolism NF-kappa B--metabolism Phosphorylation Rats Rats, Wistar Sleep Apnea, Obstructive--physiopathology Tumor Necrosis Factor-alpha--metabolism Ventricular Function, Left--physiology p38 Mitogen-Activated Protein Kinases--metabolism