Huang, Ya-Fang <br/><br/>
Redox Regulation of Pro-IL-1β Processing May Contribute to the Increased Severity of Serum-Induced Arthritis in NOX2-Deficient Mice.  [electronic resource] 

 -  Antioxidants & redox signaling  Oct 2015 
 - 973-84 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1557-7716 
<br/><br/><label>Standard No.: </label>10.1089/ars.2014.6136  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Amino Acid Chloromethyl Ketones--pharmacology<br/>Animals<br/>Ankle Joint--drug effects<br/>Arthritis--blood<br/>Caspase Inhibitors--pharmacology<br/>Cathepsin B--antagonists & inhibitors<br/>Cell Line<br/>Cytokines--metabolism<br/>Dipeptides--pharmacology<br/>Disease Models, Animal<br/>Fibroblasts--pathology<br/>Humans<br/>Inflammation--pathology<br/>Interleukin-1beta--metabolism<br/>Ketones--pharmacology<br/>Lung--cytology<br/>Membrane Glycoproteins--genetics<br/>Mice, Inbred C57BL<br/>Mice, Inbred NOD<br/>Mice, Knockout<br/>NADPH Oxidase 2<br/>NADPH Oxidases--genetics<br/>Oxidation-Reduction--drug effects<br/>Reactive Oxygen Species--metabolism<br/>Wrist Joint--drug effects