Jiang, Qian <br/><br/>
Functional loss of semaphorin 3C and/or semaphorin 3D and their epistatic interaction with ret are critical to Hirschsprung disease liability.  [electronic resource] 

 -  American journal of human genetics  Apr 2015 
 - 581-96 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1537-6605 
<br/><br/><label>Standard No.: </label>10.1016/j.ajhg.2015.02.014  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Base Sequence<br/>Epistasis, Genetic--genetics<br/>Genetic Predisposition to Disease--genetics<br/>Genetic Variation<br/>Genome-Wide Association Study<br/>Hirschsprung Disease--genetics<br/>Mice<br/>Molecular Sequence Data<br/>Proto-Oncogene Proteins c-ret--genetics<br/>Semaphorins--deficiency<br/>Sequence Analysis, DNA