Cai, Heng <br/><br/>
Roundabout4 suppresses glioma-induced endothelial cell proliferation, migration and tube formation in vitro by inhibiting VEGR2-mediated PI3K/AKT and FAK signaling pathways.  [electronic resource] 

 -  Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology  2015 
 - 1689-705 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1421-9778 
<br/><br/><label>Standard No.: </label>10.1159/000373982  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Cell Line<br/>Cell Movement--drug effects<br/>Cell Proliferation--drug effects<br/>Chromones--pharmacology<br/>Cinnamates--pharmacology<br/>Culture Media, Conditioned--pharmacology<br/>Endothelial Cells--cytology<br/>Focal Adhesion Kinase 1--antagonists & inhibitors<br/>Glioma--metabolism<br/>Humans<br/>Intercellular Signaling Peptides and Proteins--genetics<br/>Morpholines--pharmacology<br/>Neovascularization, Physiologic--drug effects<br/>Nerve Tissue Proteins--genetics<br/>Phosphatidylinositol 3-Kinases--metabolism<br/>Proto-Oncogene Proteins c-akt--antagonists & inhibitors<br/>RNA Interference<br/>Receptors, Cell Surface--antagonists & inhibitors<br/>Signal Transduction--drug effects<br/>Vascular Endothelial Growth Factor A--pharmacology<br/>Vascular Endothelial Growth Factor Receptor-2--antagonists & inhibitors<br/>Slit Homolog 2 Protein