Cannabinoid receptor agonist WIN55,212-2 and fatty acid amide hydrolase inhibitor URB597 suppress chronic cerebral hypoperfusion-induced neuronal apoptosis by inhibiting c-Jun N-terminal kinase signaling. [electronic resource]
- Neuroscience Aug 2015
- 563-75 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1873-7544
10.1016/j.neuroscience.2015.03.021 doi
Animals Apoptosis--drug effects Benzamides--therapeutic use Benzoxazines--therapeutic use Calcium Channel Blockers--therapeutic use Carbamates--therapeutic use Carotid Stenosis--drug therapy Cerebral Cortex--pathology Disease Models, Animal Dose-Response Relationship, Drug Drug Interactions Enzyme Inhibitors--therapeutic use JNK Mitogen-Activated Protein Kinases--metabolism Male Maze Learning--drug effects Morpholines--therapeutic use Naphthalenes--therapeutic use Neurons--drug effects Phosphopyruvate Hydratase--metabolism Rats Rats, Sprague-Dawley Signal Transduction--drug effects Time Factors