Zhuang, Jianguo <br/><br/>
Prenatal nicotinic exposure augments cardiorespiratory responses to activation of bronchopulmonary C-fibers.  [electronic resource] 

 -  American journal of physiology. Lung cellular and molecular physiology  May 2015 
 - L922-30 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural 
<br/><br/><label>ISSN: </label>1522-1504 
<br/><br/><label>Standard No.: </label>10.1152/ajplung.00241.2014  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Apnea--chemically induced<br/>Brain-Derived Neurotrophic Factor--biosynthesis<br/>Bronchoalveolar Lavage Fluid--chemistry<br/>Capsaicin--pharmacology<br/>Diaphragm--physiopathology<br/>Electromyography<br/>Female<br/>Ganglia--cytology<br/>Humans<br/>Hypoxia<br/>Infant, Newborn<br/>Male<br/>Methacholine Chloride--pharmacology<br/>Nerve Fibers, Unmyelinated--drug effects<br/>Nerve Growth Factor--biosynthesis<br/>Nicotine--pharmacology<br/>Nodose Ganglion--metabolism<br/>Pregnancy<br/>Prenatal Exposure Delayed Effects<br/>Random Allocation<br/>Rats<br/>Rats, Sprague-Dawley<br/>Receptor, trkA--genetics<br/>Receptor, trkB--genetics<br/>Recombinant Proteins<br/>Sensory System Agents--pharmacology<br/>Smoking--adverse effects<br/>Substance P--immunology<br/>Sudden Infant Death--etiology<br/>TRPV Cation Channels--biosynthesis