Akamata, Kaname <br/><br/>
Endothelin receptor blockade ameliorates vascular fragility in endothelial cell-specific Fli-1-knockout mice by increasing Fli-1 DNA binding ability.  [electronic resource] 

 -  Arthritis & rheumatology (Hoboken, N.J.)  May 2015 
 - 1335-44 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>2326-5205 
<br/><br/><label>Standard No.: </label>10.1002/art.39062  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Actins--metabolism<br/>Animals<br/>Bosentan<br/>Capillary Permeability<br/>Cell Line<br/>Cells, Cultured<br/>Chromatin Immunoprecipitation<br/>Endothelial Cells--metabolism<br/>Endothelin Receptor Antagonists--pharmacology<br/>Endothelin-1--metabolism<br/>Gene Expression<br/>Humans<br/>Immunoblotting<br/>Immunohistochemistry<br/>Mice<br/>Mice, Knockout<br/>Microvessels--cytology<br/>Phosphorylation<br/>Promoter Regions, Genetic<br/>Protein Kinase C-delta--metabolism<br/>Proto-Oncogene Protein c-fli-1--genetics<br/>Proto-Oncogene Proteins c-abl--metabolism<br/>RNA, Messenger--metabolism<br/>Receptors, Endothelin--metabolism<br/>Reverse Transcriptase Polymerase Chain Reaction<br/>Scleroderma, Systemic--metabolism<br/>Sulfonamides--pharmacology<br/>Vascular Diseases