Jin, Nana <br/><br/>
Truncation and activation of GSK-3β by calpain I: a molecular mechanism links to tau hyperphosphorylation in Alzheimer's disease.  [electronic resource] 

 -  Scientific reports  Feb 2015 
 - 8187 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>2045-2322 
<br/><br/><label>Standard No.: </label>10.1038/srep08187  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Alzheimer Disease--metabolism<br/>Animals<br/>Brain--drug effects<br/>Calcium--metabolism<br/>Calpain--metabolism<br/>Glycogen Synthase Kinase 3--genetics<br/>Glycogen Synthase Kinase 3 beta<br/>HEK293 Cells<br/>Humans<br/>Kainic Acid--toxicity<br/>Male<br/>Mice<br/>Phosphorylation--drug effects<br/>Proteolysis<br/>Recombinant Fusion Proteins--genetics<br/>Up-Regulation--drug effects<br/>tau Proteins--metabolism