Hoffman, Nicholas E <br/><br/>
Ca²⁺ entry via Trpm2 is essential for cardiac myocyte bioenergetics maintenance.  [electronic resource] 

 -  American journal of physiology. Heart and circulatory physiology  Mar 2015 
 - H637-50 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1522-1539 
<br/><br/><label>Standard No.: </label>10.1152/ajpheart.00720.2014  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Action Potentials<br/>Animals<br/>Calcium--metabolism<br/>Calcium Signaling<br/>Cardiomyopathies--chemically induced<br/>Disease Models, Animal<br/>Doxorubicin<br/>Energy Metabolism<br/>HEK293 Cells<br/>Humans<br/>Mice, Inbred C57BL<br/>Mice, Knockout<br/>Mitochondria, Heart--metabolism<br/>Mutation<br/>Myocardial Contraction<br/>Myocardial Reperfusion Injury--genetics<br/>Myocytes, Cardiac--metabolism<br/>Oxidative Stress<br/>Oxygen Consumption<br/>Reactive Oxygen Species--metabolism<br/>TRPM Cation Channels--deficiency<br/>Time Factors<br/>Transfection<br/>Ventricular Function, Left<br/>Ventricular Pressure