Ghosh, Swatilekha <br/><br/>
Cross-talk between endoplasmic reticulum (ER) stress and the MEK/ERK pathway potentiates apoptosis in human triple negative breast carcinoma cells: role of a dihydropyrimidone, nifetepimine.  [electronic resource] 

 -  The Journal of biological chemistry  Feb 2015 
 - 3936-49 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1083-351X 
<br/><br/><label>Standard No.: </label>10.1074/jbc.M114.594028  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Apoptosis--drug effects<br/>Blotting, Western<br/>Cell Cycle--drug effects<br/>Cell Proliferation--drug effects<br/>Endoplasmic Reticulum Chaperone BiP<br/>Endoplasmic Reticulum Stress--drug effects<br/>Female<br/>Heat-Shock Proteins--antagonists & inhibitors<br/>Humans<br/>Immunoenzyme Techniques<br/>MAP Kinase Kinase Kinases--genetics<br/>Male<br/>Mice<br/>Mice, Nude<br/>Mice, SCID<br/>Mitogen-Activated Protein Kinase 1--genetics<br/>Mitogen-Activated Protein Kinase 3--genetics<br/>Pyrimidinones--pharmacology<br/>RNA, Messenger--genetics<br/>RNA, Small Interfering--genetics<br/>Real-Time Polymerase Chain Reaction<br/>Reverse Transcriptase Polymerase Chain Reaction<br/>Triple Negative Breast Neoplasms--drug therapy<br/>Tumor Cells, Cultured<br/>Xenograft Model Antitumor Assays