Jones, Emma <br/><br/>
Hepcidin is suppressed by erythropoiesis in hemoglobin E β-thalassemia and β-thalassemia trait.  [electronic resource] 

 -  Blood  Jan 2015 
 - 873-80 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1528-0020 
<br/><br/><label>Standard No.: </label>10.1182/blood-2014-10-606491  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adolescent<br/>Adult<br/>Carrier State<br/>Case-Control Studies<br/>Child<br/>Child, Preschool<br/>Erythropoiesis--genetics<br/>Female<br/>Gene Expression Regulation<br/>Genotype<br/>Hemoglobin E--genetics<br/>Hepcidins--genetics<br/>Humans<br/>Iron--metabolism<br/>Iron Overload--etiology<br/>Linear Models<br/>Male<br/>Middle Aged<br/>Mutation<br/>Phenotype<br/>Severity of Illness Index<br/>Sri Lanka<br/>Transfusion Reaction<br/>beta-Globins--genetics<br/>beta-Thalassemia--genetics