Xu, Wei <br/><br/>
Hydration status regulates sodium flux and inflammatory pathways through epithelial sodium channel (ENaC) in the skin.  [electronic resource] 

 -  The Journal of investigative dermatology  Mar 2015 
 - 796-806 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1523-1747 
<br/><br/><label>Standard No.: </label>10.1038/jid.2014.477  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Cell Communication--physiology<br/>Cells, Cultured<br/>Cicatrix--prevention & control<br/>Coculture Techniques<br/>Cyclooxygenase 2--metabolism<br/>Cyclooxygenase 2 Inhibitors--pharmacology<br/>Dinoprostone--antagonists & inhibitors<br/>Epithelial Sodium Channels--metabolism<br/>Female<br/>Fibroblasts--metabolism<br/>Homeostasis--physiology<br/>Humans<br/>Inflammation--metabolism<br/>Keratinocytes--drug effects<br/>Models, Animal<br/>Phosphatidylinositol 3-Kinases--metabolism<br/>Proto-Oncogene Proteins c-akt--metabolism<br/>Rabbits<br/>Signal Transduction--physiology<br/>Skin--drug effects<br/>Sodium--metabolism<br/>Water--metabolism