Liu, Lin <br/><br/>
Genetic deletion of calcium/calmodulin-dependent protein kinase kinase β (CaMKK β) or CaMK IV exacerbates stroke outcomes in ovariectomized (OVXed) female mice.  [electronic resource] 

 -  BMC neuroscience  Oct 2014 
 - 118 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural 
<br/><br/><label>ISSN: </label>1471-2202 
<br/><br/><label>Standard No.: </label>10.1186/s12868-014-0118-2  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Blood-Brain Barrier--physiopathology<br/>Brain--pathology<br/>Brain Ischemia--pathology<br/>Calcium-Calmodulin-Dependent Protein Kinase Kinase--deficiency<br/>Calcium-Calmodulin-Dependent Protein Kinase Type 4--deficiency<br/>Cerebral Hemorrhage--pathology<br/>Disease Models, Animal<br/>Female<br/>Infarction, Middle Cerebral Artery<br/>Male<br/>Mice, Inbred C57BL<br/>Mice, Knockout<br/>Ovariectomy<br/>Severity of Illness Index<br/>Sex Characteristics<br/>Stroke--pathology