Rybanska-Spaeder, Ivana <br/><br/>
53BP1 mediates the fusion of mammalian telomeres rendered dysfunctional by DNA-PKcs loss or inhibition.  [electronic resource] 

 -  PloS one  2014 
 - e108731 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1932-6203 
<br/><br/><label>Standard No.: </label>10.1371/journal.pone.0108731  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adaptor Proteins, Signal Transducing<br/>Animals<br/>Ataxia Telangiectasia Mutated Proteins--metabolism<br/>Cell Cycle Proteins<br/>Chromones--pharmacology<br/>Chromosomal Proteins, Non-Histone--metabolism<br/>DNA Ligases--metabolism<br/>DNA Replication--drug effects<br/>DNA-Activated Protein Kinase--antagonists & inhibitors<br/>DNA-Binding Proteins--metabolism<br/>Embryo, Mammalian--cytology<br/>Epistasis, Genetic--drug effects<br/>Fibroblasts--drug effects<br/>G1 Phase<br/>Histones--metabolism<br/>Humans<br/>Intracellular Signaling Peptides and Proteins--metabolism<br/>Mammals--metabolism<br/>Mice<br/>Morpholines--pharmacology<br/>Poly(ADP-ribose) Polymerases--metabolism<br/>Protein Kinase Inhibitors--pharmacology<br/>S Phase--drug effects<br/>Telomere--metabolism<br/>Tumor Suppressor p53-Binding Protein 1