Shemesh, Adi <br/><br/>
Suppression of mTORC1 activation in acid-α-glucosidase-deficient cells and mice is ameliorated by leucine supplementation.  [electronic resource] 

 -  American journal of physiology. Regulatory, integrative and comparative physiology  Nov 2014 
 - R1251-9 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural 
<br/><br/><label>ISSN: </label>1522-1490 
<br/><br/><label>Standard No.: </label>10.1152/ajpregu.00212.2014  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Cell Line<br/>Dietary Supplements<br/>Dipeptides--pharmacology<br/>Disease Models, Animal<br/>Dose-Response Relationship, Drug<br/>Fibroblasts--drug effects<br/>Glycogen--metabolism<br/>Glycogen Storage Disease Type II--drug therapy<br/>Humans<br/>Insulin--pharmacology<br/>Kyphosis--enzymology<br/>Lysosomes--drug effects<br/>Mechanistic Target of Rapamycin Complex 1<br/>Mice, Inbred C57BL<br/>Mice, Knockout<br/>Motor Activity--drug effects<br/>Multiprotein Complexes--metabolism<br/>Muscle, Skeletal--drug effects<br/>Muscular Atrophy--enzymology<br/>Myoblasts--drug effects<br/>RNA Interference<br/>TOR Serine-Threonine Kinases--metabolism<br/>Transfection<br/>alpha-Glucosidases--deficiency