Li, Ping <br/><br/>
Mitotic DNA damages induced by carbon-ion radiation incur additional chromosomal breaks in polyploidy.  [electronic resource] 

 -  Toxicology letters  Oct 2014 
 - 36-47 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1879-3169 
<br/><br/><label>Standard No.: </label>10.1016/j.toxlet.2014.08.006  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Antigens, CD<br/>Apoptosis--radiation effects<br/>Cadherins--metabolism<br/>Cell Proliferation--radiation effects<br/>Checkpoint Kinase 1<br/>Chromosome Breakage<br/>DNA Damage<br/>Female<br/>G1 Phase Cell Cycle Checkpoints--radiation effects<br/>G2 Phase Cell Cycle Checkpoints--radiation effects<br/>HeLa Cells<br/>Heavy Ion Radiotherapy<br/>Histones--metabolism<br/>Humans<br/>Mitosis--radiation effects<br/>Phosphorylation<br/>Polyploidy<br/>Protein Kinases--metabolism<br/>Radiation Tolerance<br/>Time Factors<br/>Uterine Cervical Neoplasms--genetics